HEISSIG Beate, M.D., Ph.D.
Department of Research Support Utilizing Bioresource Bank
Short Biography
1984-1990 | M.D., Philipps University Marburg/ RWTH Aachen University, Germany |
1993 | Ph.D., Philipps University Marburg, Germany |
1991-1998 | Physician (Oncology, Hematology), Heidelberg/Mannheim and Tuebingen University, Germany |
1998-2003 | Senior Research Associate (Cornell University, Sloan Kettering Cancer Center), New York, USA |
2003-2004 | Assistant Professor, Juntendo University |
2004-2019 | Associate Professor, The University of Tokyo |
2019-Present | Specially Appointed Associate Professor, Juntendo University |
2019-Present | Associate Editor of Cellular Signaling; since 2020 also Associate Editor of International Journal of Molecular Sciences (IF 5.5) and American Journal of Cell Physiology (IF 4.3) |
Keywords
- Angiogenesis
- Inflammation (such as COVID-19)
- Cancer
- Protease biology
- Stem cell biology
Main Research Topics and Interests
- We seek to understand the signaling pathways involved in inflammation, cancer, and tissue repair in this department.
Publications (in English)
- Original Articles:46
- Review Articles:11
- h-Index: 31 (Web of Science)
- Sum of Times Cited: 8,933 (Web of Science)
Recent Main Publications
- Salama Y, Jaradat N, Hattori K, Heissig B. Aloysia Citrodora Essential Oil Inhibits Melanoma Cell Growth and Migration by Targeting HB-EGF-EGFR Signaling. Int J Mol Sci. Jul 29;22(15):8151, 2021 (IF 5.5)
- Salama Y, Hendrik Heida A, Yokoyama K, Takahashi S, Hattori K, Heissig B. The EGFL7-ITGB3-KLF2 axis enhances myeloma cell survival and contributes to drug resistance. Blood Advances, 24;4(6):1021-1037, 2020 (IF 5.4)
- Heissig B, Salama Y, Osada T, Takahashi S Hattori K. The multifaceted role of plasminogen in cancer and inflammation. Cellular Signaling, Nov;75:109761, 2020 (IF 4.3)
- Shimazu H, Munakata S, Tashiro Y, Salama Y, Dhahri D, Eiamboonsert S, Ota Y, Onoda H, Tsuda Y, Okada Y, Nakauchi H, Heissig B, Hattori K. Pharmacological targeting of plasmin prevents lethality and tissue damage in a murine model of macrophage activation syndrome. Blood: 6;130(1):59-72, 2017 (IF 22)
- More
Other Specific Comments
- We welcome highly motivated, competitive, self-funded postdoctoral fellows or graduate students with interest and experimental backgrounds in inflammation (including COVID-19, auto-immune diseases), stem cell biology, or cancer research.
- People joining our group should be motivated to work in an international environment and be willing to collaborate with research groups from abroad.