NIYONSABA François, M.D., Ph.D.
Department of Medical Interpreting
Short Biography
1989-1994 | M.D. China Medical University, Liaoning, China |
1995-1998 | Master’s Degree, Department of Orthopedics, China Medical University, Liaoning, China |
1999-2003 | Ph.D., Department of Host Defense and Biochemistry, Graduate School of Medicine, Juntendo University Graduate School of Medicine |
2003-2006 | Instructor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine |
2006-2007 | Assistant Professor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine |
2007-2021 | Associate Professor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine |
2010-2011 | Visiting Associate Professor, Department Microbiology and Immunology, University of British Columbia, Vancouver, Canada |
2015-2017 | Senior Associate Professor, Global Health Studies, Faculty of International Liberal Arts, Juntendo University |
2017-Present | Professor, Global Health Studies, Faculty of International Liberal Arts, Juntendo University |
2021-Present | Senior Associate Professor, Atopy (Allergy) Research Center, Juntendo University Graduate School of Medicine |
Keywords
- Antimicrobial peptides
- Infections
- Skin Barrier
- Skin Immunology
- Wound Healing
Main Research Topics and Interests
- Roles of Antimicrobial Agents in the Skin Immunity (Graduate School of Medicine)
- Global Infections (Faculty of International Liberal Arts)
Publications (in English)
- Original Articles: 62
- Review Articles: 18
- Books: 3
- h-index: 30 (Web of Science)
- Sum of Times Cited: 3,769 (Web of Science)
Recent Main Publications
- Antimicrobial peptide derived from insulin-like growth factor-binding protein 5 activates mast cells via Mas-related G protein-coupled receptor X2. Allergy 75(1): 203-207; 2020.
- Involvement of the lipoprotein receptor LRP1 in AMP-IBP5-mediated migration and proliferation of human keratinocytes and fibroblasts. J Dermatol Sci 99(3): 158-167, 2020.
- The antimicrobial peptide human β-defensin-3 accelerates wound healing by promoting angiogenesis, cell migration and proliferation through the FGFR/JAK2/STAT3 signaling pathway. Front Immunol 2021 (In press).
- More